ResearcherChinese Academy of Medical Sciences & Peking Union Medical CollegeActive in the last 2 years
Mingliang Liu
Matched on ORCID iD 0000-0001-8491-8115; OpenAlex author ID A5100616088. Sources: OpenAlex.
Their work
- Papers
- 9
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 22
2016: 1 paper · 2017: none · 2018: 3 papers · 2019: 3 papers · 2020: none · 2021: none · 2022: none · 2023: none · 2024: 2 papers
Papers 9
- Lipid-like gemcitabine diester-loaded liposomes for improved chemotherapy of pancreatic cancer. Co-author DOI 10.1016/j.jconrel.2023.11.028↗
- Improved Antitumor Efficiency of N4 -Tetradecyloxycarbonyl Gemcitabine-Loaded Liposomes for Pancreatic Cancer Chemotherapy. Co-author DOI 10.2147/ijn.s485861↗
- Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Progression. Co-author DOI 10.1158/0008-5472.can-18-0419↗
- Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization of p53 protein. Co-author DOI 10.7150/thno.30988↗
- LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. Co-author DOI 10.7150/thno.34559↗
- MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. Co-author DOI 10.1038/s41388-017-0082-2↗
- Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcription. Co-author DOI 10.1038/s41388-018-0382-1↗
- The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. Co-author DOI 10.1038/s41419-017-0158-5↗
- MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting Vimentin Expression. Co-author DOI 10.1158/1535-7163.mct-15-0877↗
Closest collaborators
The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shi-Jiang Deng · 7 shared papers, 2016–2019 · strength 0.51
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Gang Zhao · 7 shared papers, 2016–2019 · strength 0.51
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Zhu Zeng · 6 shared papers, 2016–2019 · strength 0.44
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Chunyou Wang · 6 shared papers, 2016–2019 · strength 0.42
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Guimin Xia · 2 shared papers, 2024 · strength 0.41
- Xiaowei Wang · 2 shared papers, 2024 · strength 0.41
- Ye Zeng · 5 shared papers, 2018–2019 · strength 0.39
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Shichang Deng · 5 shared papers, 2016–2019 · strength 0.34
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Shuai Zhu · 5 shared papers, 2016–2019 · strength 0.34
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Fengyu Xu · 4 shared papers, 2018–2019 · strength 0.32
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Yan Li · 1 shared paper, 2024 · strength 0.21
- Dan Wang · 1 shared paper, 2024 · strength 0.21
- Dan Wang · 1 shared paper, 2024 · strength 0.20
- Hongwei Lü · 1 shared paper, 2024 · strength 0.20
- Yan Jin · 3 shared papers, 2016–2018 · strength 0.19
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Paper MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting V… DOI 10.1158/1535-7163.mct-15-0877↗
- Xiang Li · 3 shared papers, 2016–2018 · strength 0.19
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Paper MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting V… DOI 10.1158/1535-7163.mct-15-0877↗
- Qiang Li · 2 shared papers, 2018–2019 · strength 0.15
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcrip… DOI 10.1038/s41388-018-0382-1↗
- Qiang Li · 1 shared paper, 2019 · strength 0.09
- Wei Li · 1 shared paper, 2019 · strength 0.08
- Yuhang Hu · 1 shared paper, 2019 · strength 0.08
- Qi Qin · 1 shared paper, 2018 · strength 0.08
- Yi Niu · 1 shared paper, 2018 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.