ResearcherSichuan UniversityActive in the last 2 years
Zhu Zeng
Matched on ORCID iD 0009-0004-3520-5783; OpenAlex author ID A5006411450. Sources: OpenAlex.
Their work
- Papers
- 16 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 33
2016: 1 paper · 2017: none · 2018: 2 papers · 2019: 3 papers · 2020: 1 paper · 2021: 5 papers · 2022: 1 paper · 2023: 2 papers · 2024: 1 paper
Papers 16
- tiRNA-Val-CAC-2 interacts with FUBP1 to promote pancreatic cancer metastasis by activating c‑MYC transcription. Co-author DOI 10.1038/s41388-024-02991-9↗
- NSD3, a member of nuclear receptor-binding SET domain family, is a potential prognostic biomarker for pancreatic cancer. Co-author DOI 10.1002/cam4.5774↗
- Metabolic stress-induced reciprocal loop of long noncoding RNA ZFAS1 and ZEB1 promotes epithelial-mesenchymal transition and metastasis of pancreatic cancer ce… First author DOI 10.1111/cas.15905↗
- A reciprocal feedback between N6-methyladenosine reader YTHDF3 and lncRNA DICER1-AS1 promotes glycolysis of pancreatic cancer through inhibiting maturation of … Co-author DOI 10.1186/s13046-022-02285-6↗
- Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancing glycolysis. Co-author DOI 10.1038/s41388-021-01960-w↗
- LncRNA ZNFTR functions as an inhibitor in pancreatic cancer by modulating ATF3/ZNF24/VEGFA pathway. Co-author DOI 10.1038/s41419-021-04119-3↗
- LncRNA-ZFAS1 Induces Epithelial-Mesenchymal Transition of Pancreatic Cancer Cells During Nutrient Deprivation by Promoting AMPK-Mediated Phosphorylation and St… Co-author DOI 10.21203/rs.3.rs-875697/v1↗
- Positive Reciprocal Feedback of lncRNA ZEB1-AS1 and HIF-1α Contributes to Hypoxia-Promoted Tumorigenesis and Metastasis of Pancreatic Cancer. Co-author DOI 10.3389/fonc.2021.761979↗
- Corrigendum: Positive Reciprocal Feedback of LncRNA ZEB1-AS1 and HIF-1α Contributes to Hypoxia-Promoted Tumorigenesis and Metastasis of Pancreatic Cancer. Co-author DOI 10.3389/fonc.2021.821077↗
- A reciprocal feedback of Myc and lncRNA MTSS1-AS contributes to extracellular acidity-promoted metastasis of pancreatic cancer. Co-author DOI 10.7150/thno.49147↗
- Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Progression. Co-author DOI 10.1158/0008-5472.can-18-0419↗
- Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization of p53 protein. Co-author DOI 10.7150/thno.30988↗
- LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. Co-author DOI 10.7150/thno.34559↗
- MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. Co-author DOI 10.1038/s41388-017-0082-2↗
- Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcription. Co-author DOI 10.1038/s41388-018-0382-1↗
- MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting Vimentin Expression. Co-author DOI 10.1158/1535-7163.mct-15-0877↗
Closest collaborators
The 25 strongest of 33 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Fengyu Xu · 9 shared papers, 2018–2022 · strength 0.89
- Paper A reciprocal feedback between N6-methyladenosine reader YTHDF3 and lncRNA DICER1-AS1 promotes glycolysis of pancreatic … DOI 10.1186/s13046-022-02285-6↗
- Paper LncRNA-ZFAS1 Induces Epithelial-Mesenchymal Transition of Pancreatic Cancer Cells During Nutrient Deprivation by Promot… DOI 10.21203/rs.3.rs-875697/v1↗
- Paper LncRNA ZNFTR functions as an inhibitor in pancreatic cancer by modulating ATF3/ZNF24/VEGFA pathway. DOI 10.1038/s41419-021-04119-3↗
- Gang Zhao · 10 shared papers, 2016–2021 · strength 0.87
- Paper LncRNA-ZFAS1 Induces Epithelial-Mesenchymal Transition of Pancreatic Cancer Cells During Nutrient Deprivation by Promot… DOI 10.21203/rs.3.rs-875697/v1↗
- Paper LncRNA ZNFTR functions as an inhibitor in pancreatic cancer by modulating ATF3/ZNF24/VEGFA pathway. DOI 10.1038/s41419-021-04119-3↗
- Paper Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancin… DOI 10.1038/s41388-021-01960-w↗
- Yuhang Hu · 6 shared papers, 2019–2022 · strength 0.65
- Paper A reciprocal feedback between N6-methyladenosine reader YTHDF3 and lncRNA DICER1-AS1 promotes glycolysis of pancreatic … DOI 10.1186/s13046-022-02285-6↗
- Paper LncRNA-ZFAS1 Induces Epithelial-Mesenchymal Transition of Pancreatic Cancer Cells During Nutrient Deprivation by Promot… DOI 10.21203/rs.3.rs-875697/v1↗
- Paper LncRNA ZNFTR functions as an inhibitor in pancreatic cancer by modulating ATF3/ZNF24/VEGFA pathway. DOI 10.1038/s41419-021-04119-3↗
- Chunyou Wang · 7 shared papers, 2016–2021 · strength 0.53
- Paper Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancin… DOI 10.1038/s41388-021-01960-w↗
- Paper A reciprocal feedback of Myc and lncRNA MTSS1-AS contributes to extracellular acidity-promoted metastasis of pancreatic… DOI 10.7150/thno.49147↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Ye Zeng · 6 shared papers, 2018–2021 · strength 0.49
- Paper Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancin… DOI 10.1038/s41388-021-01960-w↗
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Shi-Jiang Deng · 6 shared papers, 2016–2019 · strength 0.44
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Mingliang Liu · 6 shared papers, 2016–2019 · strength 0.44
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Shuai Zhu · 5 shared papers, 2016–2021 · strength 0.37
- Paper Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancin… DOI 10.1038/s41388-021-01960-w↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Qunli Xiong · 2 shared papers, 2023–2024 · strength 0.36
- Yongfeng Xu · 2 shared papers, 2023–2024 · strength 0.36
- Qing Zhu · 2 shared papers, 2023–2024 · strength 0.36
- Wenfeng Zhuo · 2 shared papers, 2022–2023 · strength 0.29
- Paper Metabolic stress-induced reciprocal loop of long noncoding RNA ZFAS1 and ZEB1 promotes epithelial-mesenchymal transitio… DOI 10.1111/cas.15905↗
- Paper A reciprocal feedback between N6-methyladenosine reader YTHDF3 and lncRNA DICER1-AS1 promotes glycolysis of pancreatic … DOI 10.1186/s13046-022-02285-6↗
- Yabin Xia · 2 shared papers, 2021 · strength 0.27
- Paper Positive Reciprocal Feedback of lncRNA ZEB1-AS1 and HIF-1α Contributes to Hypoxia-Promoted Tumorigenesis and Metastasis… DOI 10.3389/fonc.2021.761979↗
- Paper Corrigendum: Positive Reciprocal Feedback of LncRNA ZEB1-AS1 and HIF-1α Contributes to Hypoxia-Promoted Tumorigenesis a… DOI 10.3389/fonc.2021.821077↗
- Shichang Deng · 4 shared papers, 2016–2019 · strength 0.27
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Paper Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcrip… DOI 10.1038/s41388-018-0382-1↗
- Wei Li · 2 shared papers, 2020–2021 · strength 0.21
- Peng Xu · 2 shared papers, 2020–2021 · strength 0.19
- Paper Hypoxic exosomal HIF-1α-stabilizing circZNF91 promotes chemoresistance of normoxic pancreatic cancer cells via enhancin… DOI 10.1038/s41388-021-01960-w↗
- Paper A reciprocal feedback of Myc and lncRNA MTSS1-AS contributes to extracellular acidity-promoted metastasis of pancreatic… DOI 10.7150/thno.49147↗
- Ying Zheng · 1 shared paper, 2024 · strength 0.18
- Zhiwei Zhang · 1 shared paper, 2024 · strength 0.18
- Jinlu Liu · 1 shared paper, 2024 · strength 0.18
- Congya Zhou · 1 shared paper, 2023 · strength 0.18
- Yang Yang · 1 shared paper, 2023 · strength 0.18
- Jie Wang · 1 shared paper, 2023 · strength 0.15
- Gang Zhao · 1 shared paper, 2023 · strength 0.15
- Qiang Li · 2 shared papers, 2018–2019 · strength 0.15
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcrip… DOI 10.1038/s41388-018-0382-1↗
- Gang Zhao · 1 shared paper, 2022 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.