ResearcherWuhan Union HospitalNo recent work listed
Shi-Jiang Deng
Matched on OpenAlex author ID A5025874943. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 21
2014: 1 paper · 2015: none · 2016: 2 papers · 2017: 1 paper · 2018: 3 papers · 2019: 3 papers
Papers 10
- Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Progression. Co-author DOI 10.1158/0008-5472.can-18-0419↗
- Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization of p53 protein. Co-author DOI 10.7150/thno.30988↗
- LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. Co-author DOI 10.7150/thno.34559↗
- MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. Co-author DOI 10.1038/s41388-017-0082-2↗
- Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcription. Co-author DOI 10.1038/s41388-018-0382-1↗
- The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. Co-author DOI 10.1038/s41419-017-0158-5↗
- ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway. Co-author DOI 10.1038/cddis.2017.189↗
- MiR-548an, Transcriptionally Downregulated by HIF1α/HDAC1, Suppresses Tumorigenesis of Pancreatic Cancer by Targeting Vimentin Expression. Co-author DOI 10.1158/1535-7163.mct-15-0877↗
- Hypoxia-induced lncRNA-NUTF2P3-001 contributes to tumorigenesis of pancreatic cancer by derepressing the miR-3923/KRAS pathway. First author DOI 10.18632/oncotarget.6830↗
- Chronic pancreatitis and pancreatic cancer demonstrate active epithelial-mesenchymal transition profile, regulated by miR-217-SIRT1 pathway. Co-author DOI 10.1016/j.canlet.2014.08.007↗
Closest collaborators
The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Gang Zhao · 10 shared papers, 2014–2019 · strength 0.66
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Chunyou Wang · 9 shared papers, 2014–2019 · strength 0.56
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Mingliang Liu · 7 shared papers, 2016–2019 · strength 0.51
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Shichang Deng · 8 shared papers, 2014–2019 · strength 0.49
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Shuai Zhu · 7 shared papers, 2014–2019 · strength 0.44
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Zhu Zeng · 6 shared papers, 2016–2019 · strength 0.44
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Ye Zeng · 5 shared papers, 2018–2019 · strength 0.39
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Xiang Li · 6 shared papers, 2014–2018 · strength 0.34
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Paper ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway. DOI 10.1038/cddis.2017.189↗
- Fengyu Xu · 4 shared papers, 2018–2019 · strength 0.32
- Paper Hypoxia-induced feedback of HIF-1α and lncRNA-CF129 contributes to pancreatic cancer progression through stabilization … DOI 10.7150/thno.30988↗
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper LncRNA-MTA2TR functions as a promoter in pancreatic cancer via driving deacetylation-dependent accumulation of HIF-1α. DOI 10.7150/thno.34559↗
- Yan Jin · 5 shared papers, 2014–2018 · strength 0.29
- Paper The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. DOI 10.1038/s41419-017-0158-5↗
- Paper MiRNA-646-mediated reciprocal repression between HIF-1α and MIIP contributes to tumorigenesis of pancreatic cancer. DOI 10.1038/s41388-017-0082-2↗
- Paper ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway. DOI 10.1038/cddis.2017.189↗
- Qiang Li · 2 shared papers, 2018–2019 · strength 0.15
- Paper Nutrient Stress-Dysregulated Antisense lncRNA GLS-AS Impairs GLS-Mediated Metabolism and Represses Pancreatic Cancer Pr… DOI 10.1158/0008-5472.can-18-0419↗
- Paper Hypoxia-induced LncRNA-BX111 promotes metastasis and progression of pancreatic cancer through regulating ZEB1 transcrip… DOI 10.1038/s41388-018-0382-1↗
- Qi Qin · 2 shared papers, 2014–2018 · strength 0.11
- Qiang Li · 1 shared paper, 2019 · strength 0.09
- Wei Li · 1 shared paper, 2019 · strength 0.08
- Yuhang Hu · 1 shared paper, 2019 · strength 0.08
- Yi Niu · 1 shared paper, 2018 · strength 0.07
- Yang Liu · 1 shared paper, 2016 · strength 0.05
- Shuai Zhu · 1 shared paper, 2016 · strength 0.05
- Ming Yang · 1 shared paper, 2014 · strength 0.03
- Tao Yin · 1 shared paper, 2014 · strength 0.03
- Heshui Wu · 1 shared paper, 2014 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.