ResearcherMichael E. DeBakey VA Medical CenterActive in the last 2 years
Mien‐Chie Hung
Matched on ORCID iD 0000-0003-4317-4740; OpenAlex author ID A5076834432; NIH RePORTER profile ID 1860004. Sources: OpenAlex.
Their work
- Papers
- 18 (first author 0, last author 6)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 103
2012: 1 paper · 2013: 1 paper · 2014: none · 2015: none · 2016: 1 paper · 2017: 2 papers · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper · 2021: 2 papers · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers · 2025: none · 2026: 2 papers, 1 grant
Papers 18
- All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immunotherapy in pancreatic cancer. Last author DOI 10.1038/s41467-026-72271-5↗
- CAF-derived PAI-1 serves as an EGFR ligand and fuels KRAS-mutant pancreatic cancer. Co-author DOI 10.1038/s41467-026-75132-3↗
- Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. Co-author DOI 10.1038/s41421-024-00720-w↗
- Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. Co-author DOI 10.1038/s41467-024-47242-3↗
- All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer Last author DOI 10.1101/2024.10.20.619300↗
- Tumor-associated nonmyelinating Schwann cell-expressed PVT1 promotes pancreatic cancer kynurenine pathway and tumor immune exclusion. Co-author DOI 10.1126/sciadv.add6995↗
- Endoscopic ultrasound-guided fine-needle biopsy as a tool for studying the intra-tumoral microbiome in pancreatic ductal adenocarcinoma: a pilot study. Co-author DOI 10.1038/s41598-021-04095-w↗
- Glucocorticoid receptor regulates PD-L1 and MHC-I in pancreatic cancer cells to promote immune evasion and immunotherapy resistance. Co-author DOI 10.1038/s41467-021-27349-7↗
- Nuclear translocation of the receptor tyrosine kinase c-MET reduces the treatment efficacies of olaparib and gemcitabine in pancreatic ductal adenocarcinoma ce… Last author PMID 33520371↗
- Phase 1 trial of Vismodegib and Erlotinib combination in metastatic pancreatic cancer. Co-author DOI 10.1016/j.pan.2019.11.011↗
- Expression and Clinical Significance of Protein Kinase RNA-Like Endoplasmic Reticulum Kinase and Phosphorylated Eukaryotic Initiation Factor 2α in Pancreatic D… Co-author DOI 10.1097/mpa.0000000000001248↗
- Angiogenin/Ribonuclease 5 Is an EGFR Ligand and a Serum Biomarker for Erlotinib Sensitivity in Pancreatic Cancer. Last author DOI 10.1016/j.ccell.2018.02.012↗
- Suppression of stromal-derived Dickkopf-3 (DKK3) inhibits tumor progression and prolongs survival in pancreatic ductal adenocarcinoma. Co-author DOI 10.1126/scitranslmed.aat3487↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- A tumor vessel-targeting fusion protein elicits a chemotherapeutic bystander effect in pancreatic ductal adenocarcinoma. Last author PMID 28401019↗
- Oncogenic Functions of Gli1 in Pancreatic Adenocarcinoma Are Supported by Its PRMT1-Mediated Methylation. Last author DOI 10.1158/0008-5472.can-16-0715↗
- A tumorigenic factor interactome connected through tumor suppressor microRNA-198 in human pancreatic cancer. Co-author DOI 10.1158/1078-0432.ccr-12-3776↗
- KrasG12D-induced IKK2/β/NF-κB activation by IL-1α and p62 feedforward loops is required for development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.ccr.2011.12.006↗
Grants 1
Closest collaborators
The 25 strongest of 103 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Dihua Yu · 5 shared papers, 1 shared grant, 2021–2026 · strength 1.49
- Grant Stem Cell Biology & Cellular Therapy Program P30CA016672↗
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paul J. Chiao · 8 shared papers, 2012–2026 · strength 0.92
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Haoqiang Ying · 8 shared papers, 2017–2026 · strength 0.90
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Anirban Maitra · 7 shared papers, 2018–2026 · strength 0.87
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Huamin Wang · 9 shared papers, 2012–2026 · strength 0.83
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Elizabeth J. Shpall · 1 shared grant, 2026 · strength 0.71
- Albert C. Koong · 1 shared grant, 2026 · strength 0.71
- Kristy Brock · 1 shared grant, 2026 · strength 0.71
- Scott Kopetz · 1 shared grant, 2026 · strength 0.71
- Jianhua Ling · 6 shared papers, 2012–2026 · strength 0.65
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Jun Yao · 6 shared papers, 2017–2024 · strength 0.63
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Tumor-associated nonmyelinating Schwann cell-expressed PVT1 promotes pancreatic cancer kynurenine pathway and tumor imm… DOI 10.1126/sciadv.add6995↗
- Yongkun Wei · 5 shared papers, 2016–2026 · strength 0.61
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Jie Fu · 4 shared papers, 2017–2026 · strength 0.56
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Li‐Yuan Bai · 3 shared papers, 2022–2026 · strength 0.52
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Endoscopic ultrasound-guided fine-needle biopsy as a tool for studying the intra-tumoral microbiome in pancreatic ducta… DOI 10.1038/s41598-021-04095-w↗
- Ching-Fei Li · 3 shared papers, 2024–2026 · strength 0.51
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Ying‐Nai Wang · 3 shared papers, 2018–2026 · strength 0.42
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Angiogenin/Ribonuclease 5 Is an EGFR Ligand and a Serum Biomarker for Erlotinib Sensitivity in Pancreatic Cancer. DOI 10.1016/j.ccell.2018.02.012↗
- Jason B. Fleming · 5 shared papers, 2012–2024 · strength 0.36
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Expression and Clinical Significance of Protein Kinase RNA-Like Endoplasmic Reticulum Kinase and Phosphorylated Eukaryo… DOI 10.1097/mpa.0000000000001248↗
- Paper Angiogenin/Ribonuclease 5 Is an EGFR Ligand and a Serum Biomarker for Erlotinib Sensitivity in Pancreatic Cancer. DOI 10.1016/j.ccell.2018.02.012↗
- Liuqing Yang · 2 shared papers, 2021–2023 · strength 0.27
- Ya’an Kang · 4 shared papers, 2012–2024 · strength 0.27
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Angiogenin/Ribonuclease 5 Is an EGFR Ligand and a Serum Biomarker for Erlotinib Sensitivity in Pancreatic Cancer. DOI 10.1016/j.ccell.2018.02.012↗
- Paper Oncogenic Functions of Gli1 in Pancreatic Adenocarcinoma Are Supported by Its PRMT1-Mediated Methylation. DOI 10.1158/0008-5472.can-16-0715↗
- Ronald Anthony Depinho · 3 shared papers, 2017–2024 · strength 0.25
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Paper A tumor vessel-targeting fusion protein elicits a chemotherapeutic bystander effect in pancreatic ductal adenocarcinoma. PMID 28401019↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Rosa F. Hwang · 3 shared papers, 2016–2024 · strength 0.25
- Paper Targeting a chemo-induced adaptive signaling circuit confers therapeutic vulnerabilities in pancreatic cancer. DOI 10.1038/s41421-024-00720-w↗
- Paper Suppression of stromal-derived Dickkopf-3 (DKK3) inhibits tumor progression and prolongs survival in pancreatic ductal … DOI 10.1126/scitranslmed.aat3487↗
- Paper Oncogenic Functions of Gli1 in Pancreatic Adenocarcinoma Are Supported by Its PRMT1-Mediated Methylation. DOI 10.1158/0008-5472.can-16-0715↗
- Wenjuan Yin · 2 shared papers, 2021–2024 · strength 0.25
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Glucocorticoid receptor regulates PD-L1 and MHC-I in pancreatic cancer cells to promote immune evasion and immunotherap… DOI 10.1038/s41467-021-27349-7↗
- Xuan Zhou · 1 shared paper, 2026 · strength 0.21
- Min Li · 1 shared paper, 2026 · strength 0.21
- Bo Wang · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.