ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Jianhua Ling
Matched on ORCID iD 0000-0002-1315-4389; OpenAlex author ID A5044222908. Sources: OpenAlex.
Their work
- Papers
- 17 (first author 3, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 75
2011: 1 paper · 2012: 1 paper · 2013: none · 2014: none · 2015: none · 2016: 2 papers · 2017: 3 papers · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 3 papers · 2023: none · 2024: 2 papers · 2025: none · 2026: 1 paper
Papers 17
- All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immunotherapy in pancreatic cancer. Co-author DOI 10.1038/s41467-026-72271-5↗
- Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. First author DOI 10.1038/s41467-024-47242-3↗
- All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer Co-author DOI 10.1101/2024.10.20.619300↗
- Smad4 Deficiency Promotes Pancreatic Cancer Immunogenicity by Activating the Cancer-Autonomous DNA-Sensing Signaling Axis. Co-author DOI 10.1002/advs.202103029↗
- 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. Co-author DOI 10.1172/jci151604↗
- Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through G-protein Coupled Receptor 15. First author DOI 10.21873/anticanres.15972↗
- The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinoma. Co-author DOI 10.1111/cpr.12805↗
- Two Birds with One Stone: Therapeutic Targeting of IL1α Signaling Pathways in Pancreatic Ductal Adenocarcinoma and the Cancer-Associated Fibroblasts. Co-author DOI 10.1158/2159-8290.cd-18-1460↗
- SOX9 activity is induced by oncogenic Kras to affect MDC1 and MCMs expression in pancreatic cancer. Co-author DOI 10.1038/onc.2017.393↗
- Suppression of stromal-derived Dickkopf-3 (DKK3) inhibits tumor progression and prolongs survival in pancreatic ductal adenocarcinoma. Co-author DOI 10.1126/scitranslmed.aat3487↗
- NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. Co-author DOI 10.1016/j.canlet.2017.09.018↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- Inhibition of Pancreatic Cancer by RhIL1RA-Response. First author DOI 10.1158/1078-0432.ccr-17-0083↗
- IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. Co-author DOI 10.1158/1078-0432.ccr-14-3382↗
- Transforming Growth Factor-β Limits Secretion of Lumican by Activated Stellate Cells within Primary Pancreatic Adenocarcinoma Tumors. Co-author DOI 10.1158/1078-0432.ccr-15-2780↗
- KrasG12D-induced IKK2/β/NF-κB activation by IL-1α and p62 feedforward loops is required for development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.ccr.2011.12.006↗
- Modulation of pancreatic cancer chemoresistance by inhibition of TAK1. Co-author DOI 10.1093/jnci/djr243↗
Closest collaborators
The 25 strongest of 75 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Paul J. Chiao · 16 shared papers, 2011–2026 · strength 1.61
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Two Birds with One Stone: Therapeutic Targeting of IL1α Signaling Pathways in Pancreatic Ductal Adenocarcinoma and the … DOI 10.1158/2159-8290.cd-18-1460↗
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Jie Fu · 8 shared papers, 2016–2026 · strength 0.96
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Anirban Maitra · 5 shared papers, 2018–2026 · strength 0.69
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Huamin Wang · 7 shared papers, 2012–2026 · strength 0.65
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. DOI 10.1172/jci151604↗
- Mien‐Chie Hung · 6 shared papers, 2012–2026 · strength 0.65
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Ching-Fei Li · 3 shared papers, 2024–2026 · strength 0.51
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Lu Yu · 5 shared papers, 2017–2024 · strength 0.47
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Paper Inhibition of Pancreatic Cancer by RhIL1RA-Response. DOI 10.1158/1078-0432.ccr-17-0083↗
- Kenei Furukawa · 3 shared papers, 2020–2024 · strength 0.44
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Haoqiang Ying · 3 shared papers, 2017–2026 · strength 0.42
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Jason B. Fleming · 5 shared papers, 2012–2024 · strength 0.37
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. DOI 10.1172/jci151604↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Yongkun Wei · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Ying‐Nai Wang · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Li‐Yuan Bai · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Dihua Yu · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Ya’an Kang · 4 shared papers, 2012–2024 · strength 0.33
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. DOI 10.1172/jci151604↗
- Paper Transforming Growth Factor-β Limits Secretion of Lumican by Activated Stellate Cells within Primary Pancreatic Adenocar… DOI 10.1158/1078-0432.ccr-15-2780↗
- Peng Huang · 3 shared papers, 2017–2024 · strength 0.31
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Smad4 Deficiency Promotes Pancreatic Cancer Immunogenicity by Activating the Cancer-Autonomous DNA-Sensing Signaling Ax… DOI 10.1002/advs.202103029↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Cheng‐Wei Chou · 1 shared paper, 2026 · strength 0.21
- Wantong Yao · 1 shared paper, 2026 · strength 0.21
- Jun Yao · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Jeffrey E. Lee · 2 shared papers, 2018–2022 · strength 0.18
- Paper 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. DOI 10.1172/jci151604↗
- Paper Suppression of stromal-derived Dickkopf-3 (DKK3) inhibits tumor progression and prolongs survival in pancreatic ductal … DOI 10.1126/scitranslmed.aat3487↗
- Jie Fu · 2 shared papers, 2017–2018 · strength 0.17
- Ronald Anthony Depinho · 2 shared papers, 2017–2022 · strength 0.17
- Zhuonan Zhuang · 3 shared papers, 2016–2017 · strength 0.16
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Huai‐Qiang Ju · 3 shared papers, 2016–2017 · strength 0.16
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Matthew H. G. Katz · 2 shared papers, 2016–2022 · strength 0.16
- Paper 3D imaging analysis on an organoid-based platform guides personalized treatment in pancreatic ductal adenocarcinoma. DOI 10.1172/jci151604↗
- Paper Transforming Growth Factor-β Limits Secretion of Lumican by Activated Stellate Cells within Primary Pancreatic Adenocar… DOI 10.1158/1078-0432.ccr-15-2780↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.