ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Peng Huang
Matched on ORCID iD 0000-0002-2152-9152; OpenAlex author ID A5100644445. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 1, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 47
2015: 3 papers · 2016: none · 2017: 1 paper · 2018: none · 2019: 2 papers · 2020: 1 paper · 2021: none · 2022: 2 papers · 2023: 1 paper · 2024: 2 papers
Papers 12
- Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. Co-author DOI 10.1038/s41467-024-47242-3↗
- SQLE-mediated squalene metabolism promotes tumor immune evasion in pancreatic cancer. Last author DOI 10.3389/fimmu.2024.1512981↗
- A Cyclodiaryliodonium NOX Inhibitor for the Treatment of Pancreatic Cancer via Enzyme-Activatable Targeted Delivery by Sulfated Glycosaminoglycan Derivatives. Co-author DOI 10.1002/adhm.202203011↗
- Smad4 Deficiency Promotes Pancreatic Cancer Immunogenicity by Activating the Cancer-Autonomous DNA-Sensing Signaling Axis. Co-author DOI 10.1002/advs.202103029↗
- PLA2G2A Phospholipase Promotes Fatty Acid Synthesis and Energy Metabolism in Pancreatic Cancer Cells with K-ras Mutation. Last author DOI 10.3390/ijms231911721↗
- Oxidative stress induces monocyte-to-myofibroblast transdifferentiation through p38 in pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ctm2.41↗
- Regulation of CD137 expression through K-Ras signaling in pancreatic cancer cells. First author DOI 10.1186/s40880-019-0386-4↗
- Oncogenic K-ras Induces Mitochondrial OPA3 Expression to Promote Energy Metabolism in Pancreatic Cancer Cells. Last author DOI 10.3390/cancers12010065↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modulation. Co-author DOI 10.1158/1535-7163.mct-14-0420↗
- Selective killing of K-ras-transformed pancreatic cancer cells by targeting NAD(P)H oxidase. Co-author DOI 10.1186/s40880-015-0012-z↗
- Micro-RNA-155 is induced by K-Ras oncogenic signal and promotes ROS stress in pancreatic cancer. Last author DOI 10.18632/oncotarget.4125↗
Closest collaborators
The 25 strongest of 47 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Christophe Glorieux · 3 shared papers, 2019–2022 · strength 0.53
- Paper PLA2G2A Phospholipase Promotes Fatty Acid Synthesis and Energy Metabolism in Pancreatic Cancer Cells with K-ras Mutatio… DOI 10.3390/ijms231911721↗
- Paper Regulation of CD137 expression through K-Ras signaling in pancreatic cancer cells. DOI 10.1186/s40880-019-0386-4↗
- Paper Oncogenic K-ras Induces Mitochondrial OPA3 Expression to Promote Energy Metabolism in Pancreatic Cancer Cells. DOI 10.3390/cancers12010065↗
- Jianhua Ling · 3 shared papers, 2017–2024 · strength 0.31
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Smad4 Deficiency Promotes Pancreatic Cancer Immunogenicity by Activating the Cancer-Autonomous DNA-Sensing Signaling Ax… DOI 10.1002/advs.202103029↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paul J. Chiao · 4 shared papers, 2015–2024 · strength 0.29
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Shengping Li · 2 shared papers, 2020–2023 · strength 0.28
- Paper A Cyclodiaryliodonium NOX Inhibitor for the Treatment of Pancreatic Cancer via Enzyme-Activatable Targeted Delivery by … DOI 10.1002/adhm.202203011↗
- Paper Oxidative stress induces monocyte-to-myofibroblast transdifferentiation through p38 in pancreatic ductal adenocarcinoma. DOI 10.1002/ctm2.41↗
- Jie Fu · 3 shared papers, 2015–2024 · strength 0.25
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Junchen Pan · 1 shared paper, 2024 · strength 0.24
- Mingquan Zhang · 1 shared paper, 2022 · strength 0.22
- Huamin Wang · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Lu Yu · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Jun Yao · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Mien‐Chie Hung · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Yang Jiang · 1 shared paper, 2023 · strength 0.17
- Jiadong Pang · 1 shared paper, 2023 · strength 0.17
- Xiaobing Li · 1 shared paper, 2023 · strength 0.17
- Ping Chen · 1 shared paper, 2024 · strength 0.14
- Ching-Fei Li · 1 shared paper, 2024 · strength 0.14
- Ya’an Kang · 1 shared paper, 2024 · strength 0.14
- Qian Huang · 1 shared paper, 2024 · strength 0.14
- Kenei Furukawa · 1 shared paper, 2024 · strength 0.14
- Jun Zhao · 1 shared paper, 2024 · strength 0.14
- Wenjuan Yin · 1 shared paper, 2024 · strength 0.14
- Jason B. Fleming · 1 shared paper, 2024 · strength 0.14
- Anirban Maitra · 1 shared paper, 2024 · strength 0.14
- Ningning Niu · 1 shared paper, 2022 · strength 0.11
- Xiaojun Xia · 1 shared paper, 2022 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.