ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Lu Yu
Matched on ORCID iD 0000-0002-5216-3843; OpenAlex author ID A5100776416. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 26
2017: 3 papers · 2018: none · 2019: none · 2020: 1 paper · 2021: none · 2022: none · 2023: none · 2024: 1 paper
Papers 5
- Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. Co-author DOI 10.1038/s41467-024-47242-3↗
- The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinoma. Co-author DOI 10.1111/cpr.12805↗
- NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. Co-author DOI 10.1016/j.canlet.2017.09.018↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- Inhibition of Pancreatic Cancer by RhIL1RA-Response. Co-author DOI 10.1158/1078-0432.ccr-17-0083↗
Closest collaborators
The 25 strongest of 26 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jianhua Ling · 5 shared papers, 2017–2024 · strength 0.47
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Paper Inhibition of Pancreatic Cancer by RhIL1RA-Response. DOI 10.1158/1078-0432.ccr-17-0083↗
- Paul J. Chiao · 5 shared papers, 2017–2024 · strength 0.47
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Paper Inhibition of Pancreatic Cancer by RhIL1RA-Response. DOI 10.1158/1078-0432.ccr-17-0083↗
- Jie Fu · 4 shared papers, 2017–2024 · strength 0.38
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Kenei Furukawa · 2 shared papers, 2020–2024 · strength 0.26
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Huamin Wang · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Jun Yao · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Mien‐Chie Hung · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Peng Huang · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Ping Chen · 1 shared paper, 2024 · strength 0.14
- Ching-Fei Li · 1 shared paper, 2024 · strength 0.14
- Ya’an Kang · 1 shared paper, 2024 · strength 0.14
- Qian Huang · 1 shared paper, 2024 · strength 0.14
- Jun Zhao · 1 shared paper, 2024 · strength 0.14
- Wenjuan Yin · 1 shared paper, 2024 · strength 0.14
- Jason B. Fleming · 1 shared paper, 2024 · strength 0.14
- Anirban Maitra · 1 shared paper, 2024 · strength 0.14
- Xue Ying Qin · 1 shared paper, 2020 · strength 0.12
- Zhuonan Zhuang · 2 shared papers, 2017 · strength 0.11
- Huai‐Qiang Ju · 2 shared papers, 2017 · strength 0.11
- Yi-Chen Sun · 1 shared paper, 2017 · strength 0.09
- Jie Fu · 1 shared paper, 2017 · strength 0.09
- Lifeng Yang · 1 shared paper, 2017 · strength 0.05
- Haoqiang Ying · 1 shared paper, 2017 · strength 0.05
- Ronald Anthony Depinho · 1 shared paper, 2017 · strength 0.05
- Rui‐Hua Xu · 1 shared paper, 2017 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.