ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Huai‐Qiang Ju
Matched on ORCID iD 0000-0003-1713-5465; OpenAlex author ID A5050581018. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 22
2015: 1 paper · 2016: 3 papers · 2017: 2 papers · 2018: none · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 1 paper
Papers 7
- Corrigendum to "Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic cancer [Cancer Lett. 379 (2016) 1-11]. Co-author DOI 10.1016/j.canlet.2025.217562↗
- NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. Co-author DOI 10.1016/j.canlet.2017.09.018↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2016.05.024↗
- Melatonin overcomes gemcitabine resistance in pancreatic ductal adenocarcinoma by abrogating nuclear factor-κB activation. Co-author DOI 10.1111/jpi.12285↗
- IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. First author DOI 10.1158/1078-0432.ccr-14-3382↗
- Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modulation. Co-author DOI 10.1158/1535-7163.mct-14-0420↗
Closest collaborators
The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Paul J. Chiao · 7 shared papers, 2015–2025 · strength 0.56
- Paper Corrigendum to "Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic can… DOI 10.1016/j.canlet.2025.217562↗
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic cancer. DOI 10.1016/j.canlet.2016.05.024↗
- Rui‐Hua Xu · 4 shared papers, 2016–2025 · strength 0.40
- Paper Corrigendum to "Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic can… DOI 10.1016/j.canlet.2025.217562↗
- Paper Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic cancer. DOI 10.1016/j.canlet.2016.05.024↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Yun‐Xin Lu · 3 shared papers, 2016–2025 · strength 0.35
- Paper Corrigendum to "Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic can… DOI 10.1016/j.canlet.2025.217562↗
- Paper Regulation of the Nampt-mediated NAD salvage pathway and its therapeutic implications in pancreatic cancer. DOI 10.1016/j.canlet.2016.05.024↗
- Paper Melatonin overcomes gemcitabine resistance in pancreatic ductal adenocarcinoma by abrogating nuclear factor-κB activati… DOI 10.1111/jpi.12285↗
- Jie Fu · 4 shared papers, 2015–2017 · strength 0.21
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Zhuonan Zhuang · 3 shared papers, 2016–2017 · strength 0.16
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Jianhua Ling · 3 shared papers, 2016–2017 · strength 0.16
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Lu Yu · 2 shared papers, 2017 · strength 0.11
- Peng Huang · 2 shared papers, 2015–2017 · strength 0.10
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Lifeng Yang · 1 shared paper, 2017 · strength 0.05
- Huamin Wang · 1 shared paper, 2017 · strength 0.05
- Jun Yao · 1 shared paper, 2017 · strength 0.05
- Haoqiang Ying · 1 shared paper, 2017 · strength 0.05
- Ronald Anthony Depinho · 1 shared paper, 2017 · strength 0.05
- Mien‐Chie Hung · 1 shared paper, 2017 · strength 0.05
- Deepak Nagrath · 1 shared paper, 2017 · strength 0.05
- Wuguo Deng · 1 shared paper, 2016 · strength 0.05
- Takeshi Gocho · 1 shared paper, 2015 · strength 0.05
- Katsuhiko K. Yanaga · 1 shared paper, 2015 · strength 0.05
- Xiaoqiang Fan · 1 shared paper, 2016 · strength 0.05
- Min Li · 1 shared paper, 2016 · strength 0.05
- Jason B. Fleming · 1 shared paper, 2016 · strength 0.05
- Davide Melisi · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.