ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Jie Fu
Matched on ORCID iD 0000-0003-1794-1360; OpenAlex author ID A5100666927, A5135898681. Sources: OpenAlex.
Their work
- Papers
- 11
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 41
2015: 1 paper · 2016: 2 papers · 2017: 2 papers · 2018: none · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: none · 2024: 2 papers · 2025: none · 2026: 1 paper
Papers 11
- All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immunotherapy in pancreatic cancer. Co-author DOI 10.1038/s41467-026-72271-5↗
- Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. Co-author DOI 10.1038/s41467-024-47242-3↗
- All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer Co-author DOI 10.1101/2024.10.20.619300↗
- Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through G-protein Coupled Receptor 15. Co-author DOI 10.21873/anticanres.15972↗
- The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinoma. Co-author DOI 10.1111/cpr.12805↗
- Upregulation of the Glutaminase II Pathway Contributes to Glutamate Production upon Glutaminase 1 Inhibition in Pancreatic Cancer. Co-author DOI 10.1002/pmic.201800451↗
- NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. Co-author DOI 10.1016/j.canlet.2017.09.018↗
- Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ncomms14437↗
- Combination therapy with BPTES nanoparticles and metformin targets the metabolic heterogeneity of pancreatic cancer. Co-author DOI 10.1073/pnas.1611406113↗
- IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. Co-author DOI 10.1158/1078-0432.ccr-14-3382↗
- Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modulation. Co-author DOI 10.1158/1535-7163.mct-14-0420↗
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Paul J. Chiao · 9 shared papers, 2015–2026 · strength 1.01
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Jianhua Ling · 8 shared papers, 2016–2026 · strength 0.96
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Mien‐Chie Hung · 4 shared papers, 2017–2026 · strength 0.56
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Ching-Fei Li · 3 shared papers, 2024–2026 · strength 0.51
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Anirban Maitra · 3 shared papers, 2024–2026 · strength 0.51
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Kenei Furukawa · 3 shared papers, 2020–2024 · strength 0.44
- Paper Recombinant Thrombomodulin Has Anti-tumor Effects and Enhances the Effects of Gemcitabine for Pancreatic Cancer Through… DOI 10.21873/anticanres.15972↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Haoqiang Ying · 3 shared papers, 2017–2026 · strength 0.42
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Huamin Wang · 3 shared papers, 2017–2026 · strength 0.41
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Lu Yu · 4 shared papers, 2017–2024 · strength 0.38
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper The overexpression of long intergenic ncRNA00162 induced by RelA/p65 promotes growth of pancreatic ductal adenocarcinom… DOI 10.1111/cpr.12805↗
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Yongkun Wei · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Ying‐Nai Wang · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Li‐Yuan Bai · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Dihua Yu · 2 shared papers, 2024–2026 · strength 0.37
- Paper All-trans retinoic acid destabilizes ADAR1 protein through retinoylation-mediated USP7 dissociation and improves immuno… DOI 10.1038/s41467-026-72271-5↗
- Paper All-trans retinoic acid-mediated ADAR1 degradation synergizes with PD-1 blockade to suppress pancreatic cancer DOI 10.1101/2024.10.20.619300↗
- Peng Huang · 3 shared papers, 2015–2024 · strength 0.25
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Huai‐Qiang Ju · 4 shared papers, 2015–2017 · strength 0.21
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper Mechanisms of Overcoming Intrinsic Resistance to Gemcitabine in Pancreatic Ductal Adenocarcinoma through the Redox Modu… DOI 10.1158/1535-7163.mct-14-0420↗
- Cheng‐Wei Chou · 1 shared paper, 2026 · strength 0.21
- Wantong Yao · 1 shared paper, 2026 · strength 0.21
- Jun Yao · 2 shared papers, 2017–2024 · strength 0.20
- Paper Nardilysin-regulated scission mechanism activates polo-like kinase 3 to suppress the development of pancreatic cancer. DOI 10.1038/s41467-024-47242-3↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Jason B. Fleming · 2 shared papers, 2016–2024 · strength 0.19
- Zhuonan Zhuang · 3 shared papers, 2016–2017 · strength 0.16
- Paper NEMO peptide inhibits the growth of pancreatic ductal adenocarcinoma by blocking NF-κB activation. DOI 10.1016/j.canlet.2017.09.018↗
- Paper Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal aden… DOI 10.1038/ncomms14437↗
- Paper IL1 Receptor Antagonist Inhibits Pancreatic Cancer Growth by Abrogating NF-κB Activation. DOI 10.1158/1078-0432.ccr-14-3382↗
- Ping Chen · 1 shared paper, 2024 · strength 0.14
- Ya’an Kang · 1 shared paper, 2024 · strength 0.14
- Qian Huang · 1 shared paper, 2024 · strength 0.14
- Jun Zhao · 1 shared paper, 2024 · strength 0.14
- Wenjuan Yin · 1 shared paper, 2024 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.