ResearcherCentre National de la Recherche ScientifiqueActive in the last 2 years
Julie Guillermet‐Guibert
Matched on ORCID iD 0000-0003-3173-4907; OpenAlex author ID A5031663089. Sources: OpenAlex.
Their work
- Papers
- 15 (first author 2, last author 10)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 31
2013: 1 paper · 2014: none · 2015: 1 paper · 2016: none · 2017: 1 paper · 2018: 2 papers · 2019: 2 papers · 2020: none · 2021: 4 papers · 2022: 1 paper · 2023: none · 2024: none · 2025: 3 papers
Papers 15
- Biotechnological advances in 3D modeling of cancer initiation. Examples from pancreatic cancer research and beyond. Last author DOI 10.1088/1758-5090/adb51c↗
- Mechanical compressive forces increase PI3K output signaling in breast and pancreatic cancer cells. Last author DOI 10.26508/lsa.202402854↗
- Formation and Characterization of Two Magnetic Three-Dimensional Spheroid Models of Murine Pancreatic Adenocarcinoma. Co-author DOI 10.3390/mps8040075↗
- Upregulated Apelin Signaling in Pancreatic Cancer Activates Oncogenic Signaling Pathways to Promote Tumor Development. Co-author DOI 10.3390/ijms231810600↗
- The type of KRAS mutation drives PI3Kα/γ signalling dependency: Implication for the choice of targeted therapy in pancreatic adenocarcinoma patients. First author DOI 10.1016/j.clinre.2020.05.021↗
- Is targeting autophagy a promising lead to unveil the cloak of invisibility in pancreatic cancer? Last author DOI 10.1016/j.clinre.2021.101622↗
- Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistance. Last author DOI 10.1158/1535-7163.mct-20-0981↗
- Pancreatic cancer intrinsic PI3Kα activity accelerates metastasis and rewires macrophage component. Last author DOI 10.15252/emmm.202013502↗
- eIF4A inhibition circumvents uncontrolled DNA replication mediated by 4E-BP1 loss in pancreatic cancer. Co-author DOI 10.1172/jci.insight.121951↗
- Experimental pancreatic cancer develops in soft pancreas: novel leads for an individualized diagnosis by ultrafast elasticity imaging. Last author DOI 10.7150/thno.34066↗
- Opportunities and Resistance to Signal-targeted Therapies in Pancreatic Cancer: What Can We Learn from Proteomic Studies? Last author DOI 10.20944/preprints201802.0011.v1↗
- Signal-Targeted Therapies and Resistance Mechanisms in Pancreatic Cancer: Future Developments Reside in Proteomics. Last author DOI 10.3390/cancers10060174↗
- Attenuating PI3K isoforms in pancreatic cancer: Focus on immune PI3Kγ. First author DOI 10.1016/j.clinre.2017.04.013↗
- Implication of PI3K/Akt pathway in pancreatic cancer: When PI3K isoforms matter? Last author DOI 10.1016/j.jbior.2015.05.001↗
- Increase in PI3K signalling mimics mutated-Kras induction of pancreatic cancer Last author DOI 10.1016/j.clinre.2013.04.006↗
Closest collaborators
The 25 strongest of 31 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Nicole Therville · 6 shared papers, 2015–2025 · strength 0.70
- Paper Mechanical compressive forces increase PI3K output signaling in breast and pancreatic cancer cells. DOI 10.26508/lsa.202402854↗
- Paper Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistan… DOI 10.1158/1535-7163.mct-20-0981↗
- Paper Experimental pancreatic cancer develops in soft pancreas: novel leads for an individualized diagnosis by ultrafast elas… DOI 10.7150/thno.34066↗
- Coralie Cayron · 4 shared papers, 2021 · strength 0.70
- Paper The type of KRAS mutation drives PI3Kα/γ signalling dependency: Implication for the choice of targeted therapy in pancr… DOI 10.1016/j.clinre.2020.05.021↗
- Paper Is targeting autophagy a promising lead to unveil the cloak of invisibility in pancreatic cancer? DOI 10.1016/j.clinre.2021.101622↗
- Paper Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistan… DOI 10.1158/1535-7163.mct-20-0981↗
- Benoît Thibault · 4 shared papers, 2018–2025 · strength 0.53
- Paper Mechanical compressive forces increase PI3K output signaling in breast and pancreatic cancer cells. DOI 10.26508/lsa.202402854↗
- Paper Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistan… DOI 10.1158/1535-7163.mct-20-0981↗
- Paper Pancreatic cancer intrinsic PI3Kα activity accelerates metastasis and rewires macrophage component. DOI 10.15252/emmm.202013502↗
- Célia Cintas · 5 shared papers, 2015–2021 · strength 0.49
- Paper Opportunities and Resistance to Signal-targeted Therapies in Pancreatic Cancer: What Can We Learn from Proteomic Studie… DOI 10.20944/preprints201802.0011.v1↗
- Paper Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistan… DOI 10.1158/1535-7163.mct-20-0981↗
- Paper Pancreatic cancer intrinsic PI3Kα activity accelerates metastasis and rewires macrophage component. DOI 10.15252/emmm.202013502↗
- Céline Basset · 4 shared papers, 2017–2021 · strength 0.43
- Paper Attenuating PI3K isoforms in pancreatic cancer: Focus on immune PI3Kγ. DOI 10.1016/j.clinre.2017.04.013↗
- Paper Experimental pancreatic cancer develops in soft pancreas: novel leads for an individualized diagnosis by ultrafast elas… DOI 10.7150/thno.34066↗
- Paper Pancreatic cancer intrinsic PI3Kα activity accelerates metastasis and rewires macrophage component. DOI 10.15252/emmm.202013502↗
- Romain Baer · 4 shared papers, 2013–2021 · strength 0.30
- Paper Pancreatic cancer intrinsic PI3Kα activity accelerates metastasis and rewires macrophage component. DOI 10.15252/emmm.202013502↗
- Paper eIF4A inhibition circumvents uncontrolled DNA replication mediated by 4E-BP1 loss in pancreatic cancer. DOI 10.1172/jci.insight.121951↗
- Paper Implication of PI3K/Akt pathway in pancreatic cancer: When PI3K isoforms matter? DOI 10.1016/j.jbior.2015.05.001↗
- Maxime Lafond · 1 shared paper, 2025 · strength 0.28
- Cyril Lafon · 1 shared paper, 2025 · strength 0.28
- Magali Perier · 1 shared paper, 2025 · strength 0.28
- Charles Handschin · 1 shared paper, 2025 · strength 0.27
- Mickaël Di‐Luoffo · 1 shared paper, 2025 · strength 0.25
- Pierre Cordelier · 2 shared papers, 2021–2022 · strength 0.21
- Stéphane Pyronnet · 2 shared papers, 2013–2019 · strength 0.14
- Carline Chaves-Almagro · 1 shared paper, 2022 · strength 0.12
- Bernard Masri · 1 shared paper, 2022 · strength 0.12
- Audrey Francès · 1 shared paper, 2022 · strength 0.12
- Zahra Dantes · 1 shared paper, 2021 · strength 0.10
- Paola Cappello · 1 shared paper, 2021 · strength 0.10
- Maximilian Reichert · 1 shared paper, 2021 · strength 0.10
- Fabrice Muscari · 1 shared paper, 2021 · strength 0.09
- Chiara Falcomatà · 1 shared paper, 2021 · strength 0.09
- Gabriela Reyes‐Castellanos · 1 shared paper, 2021 · strength 0.09
- Barbara Bournet · 1 shared paper, 2021 · strength 0.09
- Alice Carrier · 1 shared paper, 2021 · strength 0.09
- Dieter Saur · 1 shared paper, 2021 · strength 0.09
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.