ResearcherUniversity of LiverpoolActive in the last 2 years
Valeria Quaranta
Matched on OpenAlex author ID A5019450534. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 17
2016: 3 papers · 2017: none · 2018: 2 papers · 2019: none · 2020: none · 2021: none · 2022: 1 paper · 2023: none · 2024: 3 papers
Papers 9
- Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. Co-author DOI 10.1038/s41467-024-47949-3↗
- Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Co-author DOI 10.1038/s43018-024-00731-2↗
- Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Co-author DOI 10.1038/s43018-024-00751-y↗
- Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. Co-author DOI 10.1136/gutjnl-2021-325272↗
- Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-17-3876↗
- Macrophages suppress anti-tumour immunity in metastatic pancreatic cancer Co-author DOI 10.17638/03032847↗
- Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. First author DOI 10.1038/ncb3340↗
- Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. First author DOI 10.1038/ncb3377↗
- Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. Co-author DOI 10.1158/0008-5472.can-16-1201↗
Closest collaborators
The 17 strongest of 17 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Michael C. Schmid · 8 shared papers, 2016–2024 · strength 0.85
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Ainhoa Mielgo · 8 shared papers, 2016–2024 · strength 0.85
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Daniel H. Palmer · 7 shared papers, 2016–2024 · strength 0.81
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Meirion Raymant · 5 shared papers, 2018–2024 · strength 0.71
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Yuliana Astuti · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Paula Ghaneh · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Gaia Bellomo · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Jennifer P. Morton · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Christopher Michael Halloran · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Robert Peter Jones · 3 shared papers, 2022–2024 · strength 0.47
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Carolyn Rainer · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Sebastian Rune Nielsen · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Dannielle D. Engle · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- David Arthur Tuveson · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Mark A. Glenn · 1 shared paper, 2024 · strength 0.18
- Lucy Ireland · 3 shared papers, 2016 · strength 0.14
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Paper Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3377↗
- Pedro Antonio Pérez-Mancera · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.