ResearcherUniversity of LiverpoolActive in the last 2 years
Ainhoa Mielgo
Matched on ORCID iD 0000-0002-4159-5931; OpenAlex author ID A5024297597. Sources: OpenAlex.
Their work
- Papers
- 14 (first author 0, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 23
2013: 1 paper · 2014: none · 2015: none · 2016: 3 papers · 2017: none · 2018: 1 paper · 2019: none · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: none · 2024: 6 papers · 2025: 1 paper
Papers 14
- Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: meta-analysis. Co-author DOI 10.1093/bjsopen/zrae172↗
- Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. Last author DOI 10.1016/j.pan.2024.04.008↗
- Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. Co-author DOI 10.1038/s41467-024-47949-3↗
- Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Co-author DOI 10.1038/s43018-024-00731-2↗
- Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Co-author DOI 10.1038/s43018-024-00751-y↗
- Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. Last author DOI 10.1158/0008-5472.can-23-1542↗
- Robust and consistent biomarker candidates identification by a machine learning approach applied to pancreatic ductal adenocarcinoma metastasis. Co-author DOI 10.1186/s12911-024-02578-0↗
- Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. Co-author DOI 10.1136/gutjnl-2021-325272↗
- Blockade of Stromal Gas6 Alters Cancer Cell Plasticity, Activates NK Cells, and Inhibits Pancreatic Cancer Metastasis. Last author DOI 10.3389/fimmu.2020.00297↗
- Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-17-3876↗
- Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3340↗
- Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3377↗
- Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. Last author DOI 10.1158/0008-5472.can-16-1201↗
- Impact of tumour associated macrophages in pancreatic cancer. Co-author DOI 10.5483/bmbrep.2013.46.3.036↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Michael C. Schmid · 13 shared papers, 2013–2025 · strength 1.83
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paula Ghaneh · 6 shared papers, 2022–2025 · strength 1.10
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Christopher Michael Halloran · 6 shared papers, 2022–2025 · strength 1.10
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Meirion Raymant · 6 shared papers, 2018–2024 · strength 0.99
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Valeria Quaranta · 8 shared papers, 2016–2024 · strength 0.85
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Gaia Bellomo · 5 shared papers, 2022–2024 · strength 0.84
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Daniel H. Palmer · 7 shared papers, 2016–2024 · strength 0.81
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Robert Peter Jones · 4 shared papers, 2022–2024 · strength 0.67
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Mark A. Glenn · 3 shared papers, 2024 · strength 0.66
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Teifion Luckett · 3 shared papers, 2020–2024 · strength 0.64
- Paper Robust and consistent biomarker candidates identification by a machine learning approach applied to pancreatic ductal a… DOI 10.1186/s12911-024-02578-0↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Blockade of Stromal Gas6 Alters Cancer Cell Plasticity, Activates NK Cells, and Inhibits Pancreatic Cancer Metastasis. DOI 10.3389/fimmu.2020.00297↗
- Yuliana Astuti · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Jennifer P. Morton · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Lucy Ireland · 5 shared papers, 2016–2024 · strength 0.50
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Blockade of Stromal Gas6 Alters Cancer Cell Plasticity, Activates NK Cells, and Inhibits Pancreatic Cancer Metastasis. DOI 10.3389/fimmu.2020.00297↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Carolyn Rainer · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Sufyan Elhashani · 1 shared paper, 2024 · strength 0.28
- Tanakamol Mahawan · 1 shared paper, 2024 · strength 0.28
- Eva Caamaño‐Gutiérrez · 1 shared paper, 2024 · strength 0.28
- Luke Dickerson · 1 shared paper, 2025 · strength 0.26
- Daniel H. Palmer · 1 shared paper, 2025 · strength 0.26
- Sebastian Rune Nielsen · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Dannielle D. Engle · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- David Arthur Tuveson · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Pedro Antonio Pérez-Mancera · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.