ResearcherUniversity of CopenhagenActive in the last 2 years
Sebastian Rune Nielsen
Matched on ORCID iD 0000-0002-8449-476X; OpenAlex author ID A5043813099. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 19
2016: 3 papers · 2017: none · 2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper · 2022: none · 2023: 1 paper · 2024: none · 2025: none · 2026: 1 paper
Papers 7
- A serpin-myeloid axis in pancreatic cancer heterogeneity and immune evasion. Co-author DOI 10.1038/s41586-026-11002-8↗
- FES null mice demonstrate a reduction in neutrophil dependent pancreatic cancer metastatic burden. Co-author DOI 10.3389/fonc.2023.1096499↗
- Suppression of tumor-associated neutrophils by lorlatinib attenuates pancreatic cancer growth and improves treatment with immune checkpoint blockade. Co-author DOI 10.1038/s41467-021-23731-7↗
- Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-17-3876↗
- Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3340↗
- Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3377↗
- Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. Co-author DOI 10.1158/0008-5472.can-16-1201↗
Closest collaborators
The 19 strongest of 19 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Janine Terra Erler · 2 shared papers, 2021–2023 · strength 0.33
- Jan E. Strøbech · 2 shared papers, 2021–2023 · strength 0.33
- David Arthur Tuveson · 5 shared papers, 2016–2021 · strength 0.32
- Paper Suppression of tumor-associated neutrophils by lorlatinib attenuates pancreatic cancer growth and improves treatment wi… DOI 10.1038/s41467-021-23731-7↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Miriam Merad · 1 shared paper, 2026 · strength 0.26
- Chiara Falcomatà · 1 shared paper, 2026 · strength 0.26
- Brian D Brown · 1 shared paper, 2026 · strength 0.26
- Valeria Quaranta · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Carolyn Rainer · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Michael C. Schmid · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Ainhoa Mielgo Iza · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Dannielle D. Engle · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Daniel H. Palmer · 3 shared papers, 2016–2018 · strength 0.16
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Paper Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3377↗
- Lucy Ireland · 3 shared papers, 2016 · strength 0.14
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Paper Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3377↗
- Chang‐Il Hwang · 1 shared paper, 2021 · strength 0.11
- Luís Arnes · 1 shared paper, 2021 · strength 0.11
- Jennifer P. Morton · 1 shared paper, 2021 · strength 0.11
- Owen James Sansom · 1 shared paper, 2021 · strength 0.11
- Meirion Raymant Raymant · 1 shared paper, 2018 · strength 0.07
- Pedro Antonio Pérez-Mancera · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.