ResearcherUniversity of LiverpoolNo recent work listed
Carolyn Rainer
Matched on OpenAlex author ID A5033849863. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 16
2016: 3 papers · 2017: none · 2018: 1 paper · 2019: none · 2020: 1 paper · 2021: none · 2022: 1 paper
Papers 6
- Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. First author DOI 10.1136/gutjnl-2021-325272↗
- Investigating the response of the hepatic metastatic niche to chemotherapy in pancreatic cancer Co-author DOI 10.17638/03121678↗
- Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. First author DOI 10.1158/0008-5472.can-17-3876↗
- Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3340↗
- Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Co-author DOI 10.1038/ncb3377↗
- Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. Co-author DOI 10.1158/0008-5472.can-16-1201↗
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Valeria Quaranta · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Michael C. Schmid · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Ainhoa Mielgo Iza · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Daniel H. Palmer · 4 shared papers, 2016–2022 · strength 0.29
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Sebastian Rune Nielsen · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Dannielle D. Engle · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- David Arthur Tuveson · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Meirion Raymant Raymant · 2 shared papers, 2018–2022 · strength 0.20
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Lucy Ireland · 3 shared papers, 2016 · strength 0.14
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Paper Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3377↗
- Yuliana Astuti · 1 shared paper, 2022 · strength 0.13
- Robert Peter Jones · 1 shared paper, 2022 · strength 0.13
- Paula Ghaneh · 1 shared paper, 2022 · strength 0.13
- Gaia Bellomo · 1 shared paper, 2022 · strength 0.13
- Jennifer P. Morton · 1 shared paper, 2022 · strength 0.13
- Christopher Michael Halloran · 1 shared paper, 2022 · strength 0.13
- Pedro Antonio Pérez-Mancera · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.