ResearcherUniversity of LiverpoolActive in the last 2 years
Michael C. Schmid
Matched on ORCID iD 0000-0002-3445-0013; OpenAlex author ID A5056700264. Sources: OpenAlex.
Their work
- Papers
- 17 (first author 1, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 44
2013: 1 paper · 2014: none · 2015: none · 2016: 5 papers · 2017: none · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: none · 2024: 5 papers · 2025: 1 paper
Papers 17
- Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: meta-analysis. Co-author DOI 10.1093/bjsopen/zrae172↗
- Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. Co-author DOI 10.1016/j.pan.2024.04.008↗
- Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. Last author DOI 10.1038/s41467-024-47949-3↗
- Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Last author DOI 10.1038/s43018-024-00731-2↗
- Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. Last author DOI 10.1038/s43018-024-00751-y↗
- Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. Co-author DOI 10.1158/0008-5472.can-23-1542↗
- Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. Last author DOI 10.1136/gutjnl-2021-325272↗
- Blockade of Stromal Gas6 Alters Cancer Cell Plasticity, Activates NK Cells, and Inhibits Pancreatic Cancer Metastasis. Co-author DOI 10.3389/fimmu.2020.00297↗
- MST1R kinase accelerates pancreatic cancer progression via effects on both epithelial cells and macrophages. Co-author DOI 10.1038/s41388-019-0811-9↗
- Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. Last author DOI 10.1158/0008-5472.can-17-3876↗
- F1F0-ATP Synthase Inhibitory Factor 1 in the Normal Pancreas and in Pancreatic Ductal Adenocarcinoma: Effects on Bioenergetics, Invasion and Proliferation. Co-author DOI 10.3389/fphys.2018.00833↗
- UHRF1 regulation of the Keap1-Nrf2 pathway in pancreatic cancer contributes to oncogenesis. Co-author DOI 10.1002/path.4665↗
- Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Last author DOI 10.1038/ncb3340↗
- Corrigendum: Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. Last author DOI 10.1038/ncb3377↗
- Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. Co-author DOI 10.1158/0008-5472.can-16-1201↗
- Macrophage PI3Kγ Drives Pancreatic Ductal Adenocarcinoma Progression. Co-author DOI 10.1158/2159-8290.cd-15-1346↗
- Impact of tumour associated macrophages in pancreatic cancer. First author DOI 10.5483/bmbrep.2013.46.3.036↗
Closest collaborators
The 25 strongest of 44 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Ainhoa Mielgo Iza · 13 shared papers, 2013–2025 · strength 1.83
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paula Ghaneh · 6 shared papers, 2022–2025 · strength 1.10
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Christopher Michael Halloran · 6 shared papers, 2022–2025 · strength 1.10
- Paper Neoadjuvant treatment versus upfront surgery in borderline resectable and resectable pancreatic ductal adenocarcinoma: … DOI 10.1093/bjsopen/zrae172↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Meirion Raymant Raymant · 6 shared papers, 2018–2024 · strength 0.99
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Valeria Quaranta · 8 shared papers, 2016–2024 · strength 0.85
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Gaia Bellomo · 5 shared papers, 2022–2024 · strength 0.84
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Daniel H. Palmer · 7 shared papers, 2016–2024 · strength 0.81
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Robert Peter Jones · 4 shared papers, 2022–2024 · strength 0.67
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Mark A. Glenn · 3 shared papers, 2024 · strength 0.66
- Paper Expression of versican isoforms V0/V1 by pancreatic cancer associated fibroblasts increases fibroblast proliferation. DOI 10.1016/j.pan.2024.04.008↗
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Yuliana Astuti · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Jennifer P. Morton · 4 shared papers, 2022–2024 · strength 0.64
- Paper Macrophage-fibroblast JAK/STAT dependent crosstalk promotes liver metastatic outgrowth in pancreatic cancer. DOI 10.1038/s41467-024-47949-3↗
- Paper Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00731-2↗
- Paper Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis. DOI 10.1038/s43018-024-00751-y↗
- Lucy Ireland · 5 shared papers, 2016–2024 · strength 0.50
- Paper Mesothelin Secretion by Pancreatic Cancer Cells Co-opts Macrophages and Promotes Metastasis. DOI 10.1158/0008-5472.can-23-1542↗
- Paper Blockade of Stromal Gas6 Alters Cancer Cell Plasticity, Activates NK Cells, and Inhibits Pancreatic Cancer Metastasis. DOI 10.3389/fimmu.2020.00297↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Teifion Luckett · 2 shared papers, 2020–2024 · strength 0.36
- Carolyn Rainer · 5 shared papers, 2016–2022 · strength 0.34
- Paper Chemotherapy-induced infiltration of neutrophils promotes pancreatic cancer metastasis via Gas6/AXL signalling axis. DOI 10.1136/gutjnl-2021-325272↗
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Sufyan Elhashani · 1 shared paper, 2024 · strength 0.28
- Luke Dickerson · 1 shared paper, 2025 · strength 0.26
- Daniel H. Palmer · 1 shared paper, 2025 · strength 0.26
- Sebastian Rune Nielsen · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Dannielle D. Engle · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- David Arthur Tuveson · 4 shared papers, 2016–2018 · strength 0.21
- Paper Macrophage-Derived Granulin Drives Resistance to Immune Checkpoint Inhibition in Metastatic Pancreatic Cancer. DOI 10.1158/0008-5472.can-17-3876↗
- Paper Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors. DOI 10.1158/0008-5472.can-16-1201↗
- Paper Macrophage-secreted granulin supports pancreatic cancer metastasis by inducing liver fibrosis. DOI 10.1038/ncb3340↗
- Eithne Costello · 2 shared papers, 2016–2018 · strength 0.12
- Philippe Foubert · 2 shared papers, 2016–2019 · strength 0.12
- Judith A. Varner · 2 shared papers, 2016–2019 · strength 0.12
- Andrew M. Lowy · 2 shared papers, 2016–2019 · strength 0.12
- Helen Tanton · 1 shared paper, 2018 · strength 0.08
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.