ResearcherUniversity of VeronaActive in the last 2 years
Antonia Malinova
Matched on ORCID iD 0000-0003-0325-501X; OpenAlex author ID A5058685554. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 35
2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 1 paper
Papers 5
- MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. First author DOI 10.1038/s41586-025-08721-9↗
- Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. Co-author DOI 10.1136/gutjnl-2023-329807↗
- LAMC2 Regulates Key Transcriptional and Targetable Effectors to Support Pancreatic Cancer Growth. Co-author DOI 10.1158/1078-0432.ccr-22-0794↗
- Loss of FGFR4 promotes the malignant phenotype of PDAC. Co-author DOI 10.1038/s41388-022-02432-5↗
- Cell Lineage Infidelity in PDAC Progression and Therapy Resistance. Co-author DOI 10.3389/fcell.2021.795251↗
Closest collaborators
The 25 strongest of 35 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Vincenzo Corbo · 5 shared papers, 2021–2025 · strength 0.76
- Paper Cell Lineage Infidelity in PDAC Progression and Therapy Resistance. DOI 10.3389/fcell.2021.795251↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Lisa Veghini · 4 shared papers, 2021–2025 · strength 0.62
- Paper Cell Lineage Infidelity in PDAC Progression and Therapy Resistance. DOI 10.3389/fcell.2021.795251↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Aldo Scarpa Scarpa · 4 shared papers, 2022–2025 · strength 0.57
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper LAMC2 Regulates Key Transcriptional and Targetable Effectors to Support Pancreatic Cancer Growth. DOI 10.1158/1078-0432.ccr-22-0794↗
- Francesca Lupo · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Peter J. Bailey · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Sabrina L. D’Agosto · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Rita Teresa Lawlor · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Elena Fiorini · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Pietro Delfino · 3 shared papers, 2022–2024 · strength 0.40
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper LAMC2 Regulates Key Transcriptional and Targetable Effectors to Support Pancreatic Cancer Growth. DOI 10.1158/1078-0432.ccr-22-0794↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Carmine Carbone · 2 shared papers, 2024–2025 · strength 0.32
- David Arthur Tuveson · 2 shared papers, 2024–2025 · strength 0.32
- Claudio Luchini · 2 shared papers, 2022–2025 · strength 0.28
- Michèle Milella · 2 shared papers, 2022–2025 · strength 0.28
- Francisco X. Real · 1 shared paper, 2021 · strength 0.19
- Giuseppe Malleo · 1 shared paper, 2025 · strength 0.17
- Antonio Agostini · 1 shared paper, 2025 · strength 0.17
- Daniel Schreyer · 1 shared paper, 2025 · strength 0.17
- Christian P. Pilarsky · 1 shared paper, 2025 · strength 0.17
- Roberto Salvia · 1 shared paper, 2025 · strength 0.17
- Vincenzo Bronte · 1 shared paper, 2024 · strength 0.15
- Francesco De Sanctis · 1 shared paper, 2024 · strength 0.15
- Ivano Amelio · 1 shared paper, 2024 · strength 0.15
- Geny Piro · 1 shared paper, 2024 · strength 0.15
- Chang‐Il Hwang · 1 shared paper, 2024 · strength 0.15
- Rodrigo Entrialgo‐Cadierno · 1 shared paper, 2023 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.