ResearcherChampalimaud FoundationActive in the last 2 years
Peter J. Bailey
Matched on ORCID iD 0000-0002-0857-2041; OpenAlex author ID A5102937267. Sources: OpenAlex.
Their work
- Papers
- 42 (first author 7, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 191
2014: 1 paper · 2015: 2 papers · 2016: 4 papers · 2017: 5 papers · 2018: 1 paper · 2019: 4 papers · 2020: 3 papers · 2021: 6 papers · 2022: 4 papers · 2023: 3 papers · 2024: 5 papers · 2025: 2 papers · 2026: 2 papers
Papers 42
- Pancreatic Cancer Frequently Infiltrates the TRIANGLE Compartment: A Surgical Option to Prevent Local Recurrence. Co-author DOI 10.1097/as9.0000000000000672↗
- The Role of Circulating Tumor DNA in Surgical Management of Pancreatic Cancer: Systematic Review and Meta-analysis. Co-author DOI 10.1097/sla.0000000000007036↗
- MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. Co-author DOI 10.1038/s41586-025-08721-9↗
- Pancreatic Adenocarcinoma: Long-Term Outcomes of Adjuvant Therapy in the ESPAC4 Phase III Trial. Co-author DOI 10.1200/jco.24.01118↗
- Expression of the membrane tetraspanin claudin 18 on cancer cells promotes T lymphocyte infiltration and antitumor immunity in pancreatic cancer. Co-author DOI 10.1016/j.immuni.2024.04.021↗
- Metavert synergises with standard cytotoxics in human PDAC organoids and is associated with transcriptomic signatures of therapeutic response. Co-author DOI 10.1016/j.tranon.2024.102109↗
- Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. Co-author DOI 10.1136/gutjnl-2023-329807↗
- Tumour plasticity and tumour microenvironment interactions as potential immunologic targets for pancreatic cancer treatment. Co-author DOI 10.21037/cco-24-72↗
- ESMO 2023 pancreatic cancer guidelines signal stepwise progress. First author DOI 10.21037/hbsn-24-37↗
- Persister cell phenotypes contribute to poor patient outcomes after neoadjuvant chemotherapy in PDAC. Last author DOI 10.1038/s43018-023-00628-6↗
- Refining the Treatment of Pancreatic Cancer From Big Data to Improved Individual Survival. Co-author DOI 10.1093/function/zqad011↗
- Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. Co-author DOI 10.3389/fcell.2023.1179559↗
- Loss of FGFR4 promotes the malignant phenotype of PDAC. Co-author DOI 10.1038/s41388-022-02432-5↗
- CDK7 inhibition augments response to multidrug chemotherapy in pancreatic cancer. Co-author DOI 10.1186/s13046-022-02443-w↗
- Modulation of Type I Interferon Responses to Influence Tumor-Immune Cross Talk in PDAC. First author DOI 10.3389/fcell.2022.816517↗
- Editorial: Translational Insights Into Pancreatic Ductal Adenocarcinoma. First author DOI 10.3389/fcell.2022.875836↗
- Poly(ADP-ribose) polymerase inhibition in pancreatic cancer. First author DOI 10.1002/gcc.22932↗
- Targeting DNA Damage Response and Replication Stress in Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2020.09.043↗
- Perioperative immunotherapy for pancreatic cancer is on its way. First author DOI 10.21037/hbsn-21-238↗
- Clinical Impact of Molecular Subtyping of Pancreatic Cancer. Co-author DOI 10.3389/fcell.2021.743908↗
- Deconstructing Pancreatic Cancer Using Next Generation-Omic Technologies-From Discovery to Knowledge-Guided Platforms for Better Patient Management. Last author DOI 10.3389/fcell.2021.795735↗
- Deep Learning Improves Pancreatic Cancer Diagnosis Using RNA-Based Variants. Co-author DOI 10.3390/cancers13112654↗
- Altered RNA Splicing by Mutant p53 Activates Oncogenic RAS Signaling in Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2020.05.010↗
- Using Chromatin Accessibility to Delineate Therapeutic Subtypes in Pancreatic Cancer Patient-Derived Cell Lines. Last author DOI 10.1016/j.xpro.2020.100079↗
- Precision Oncology in Surgery: Patient Selection for Operable Pancreatic Cancer. Co-author DOI 10.1097/sla.0000000000003143↗
- The integrin αvβ6 drives pancreatic cancer through diverse mechanisms and represents an effective target for therapy. Co-author DOI 10.1002/path.5320↗
- Molecular subtypes of pancreatic cancer. First author DOI 10.1038/s41575-019-0109-y↗
- Prolactin Promotes Fibrosis and Pancreatic Cancer Progression. Co-author DOI 10.1158/0008-5472.can-18-3064↗
- CRISPR Cas9 in Pancreatic Cancer Research. First author DOI 10.3389/fcell.2019.00239↗
- ADAM12 is a circulating marker for stromal activation in pancreatic cancer and predicts response to chemotherapy. Co-author DOI 10.1038/s41389-018-0096-9↗
- BRCA2 secondary mutation-mediated resistance to platinum and PARP inhibitor-based therapy in pancreatic cancer. Co-author DOI 10.1038/bjc.2017.40↗
- An integrative approach unveils FOSL1 as an oncogene vulnerability in KRAS-driven lung and pancreatic cancer. Co-author DOI 10.1038/ncomms14294↗
- Recurrent noncoding regulatory mutations in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/ng.3861↗
- Hypermutation In Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2016.09.060↗
- Pancreatic Cancer Genomes: Implications for Clinical Management and Therapeutic Development. Co-author DOI 10.1158/1078-0432.ccr-16-2411↗
- CXCR2 Inhibition Profoundly Suppresses Metastases and Augments Immunotherapy in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1016/j.ccell.2016.04.014↗
- Genomic analyses identify molecular subtypes of pancreatic cancer. Co-author DOI 10.1038/nature16965↗
- Exploiting the neoantigen landscape for immunotherapy of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/srep35848↗
- PDX1 dynamically regulates pancreatic ductal adenocarcinoma initiation and maintenance. Co-author DOI 10.1101/gad.291021.116↗
- Whole genomes redefine the mutational landscape of pancreatic cancer. Co-author DOI 10.1038/nature14169↗
- Targeting the LOX/hypoxia axis reverses many of the features that make pancreatic cancer deadly: inhibition of LOX abrogates metastasis and enhances drug effic… Co-author DOI 10.15252/emmm.201404827↗
- Genome-wide DNA methylation patterns in pancreatic ductal adenocarcinoma reveal epigenetic deregulation of SLIT-ROBO, ITGA2 and MET signaling. Co-author DOI 10.1002/ijc.28765↗
Closest collaborators
The 25 strongest of 191 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- John P. Neoptolemos · 11 shared papers, 2021–2025 · strength 1.97
- Paper ESMO 2023 pancreatic cancer guidelines signal stepwise progress. DOI 10.21037/hbsn-24-37↗
- Paper Tumour plasticity and tumour microenvironment interactions as potential immunologic targets for pancreatic cancer treat… DOI 10.21037/cco-24-72↗
- Paper Refining the Treatment of Pancreatic Cancer From Big Data to Improved Individual Survival. DOI 10.1093/function/zqad011↗
- Christoph Springfeld · 10 shared papers, 2021–2025 · strength 1.78
- Paper ESMO 2023 pancreatic cancer guidelines signal stepwise progress. DOI 10.21037/hbsn-24-37↗
- Paper Tumour plasticity and tumour microenvironment interactions as potential immunologic targets for pancreatic cancer treat… DOI 10.21037/cco-24-72↗
- Paper Refining the Treatment of Pancreatic Cancer From Big Data to Improved Individual Survival. DOI 10.1093/function/zqad011↗
- Andrew Victor Biankin · 16 shared papers, 2014–2022 · strength 1.10
- Paper Molecular subtypes of pancreatic cancer. DOI 10.1038/s41575-019-0109-y↗
- Paper CDK7 inhibition augments response to multidrug chemotherapy in pancreatic cancer. DOI 10.1186/s13046-022-02443-w↗
- Paper Pancreatic Cancer Genomes: Implications for Clinical Management and Therapeutic Development. DOI 10.1158/1078-0432.ccr-16-2411↗
- Markus Wolfgang Büchler · 6 shared papers, 2021–2026 · strength 1.08
- Paper The Role of Circulating Tumor DNA in Surgical Management of Pancreatic Cancer: Systematic Review and Meta-analysis. DOI 10.1097/sla.0000000000007036↗
- Paper Pancreatic Cancer Frequently Infiltrates the TRIANGLE Compartment: A Surgical Option to Prevent Local Recurrence. DOI 10.1097/as9.0000000000000672↗
- Paper Refining the Treatment of Pancreatic Cancer From Big Data to Improved Individual Survival. DOI 10.1093/function/zqad011↗
- David Kuang-Fu Chang · 15 shared papers, 2014–2022 · strength 1.02
- Paper Molecular subtypes of pancreatic cancer. DOI 10.1038/s41575-019-0109-y↗
- Paper CDK7 inhibition augments response to multidrug chemotherapy in pancreatic cancer. DOI 10.1186/s13046-022-02443-w↗
- Paper Pancreatic Cancer Genomes: Implications for Clinical Management and Therapeutic Development. DOI 10.1158/1078-0432.ccr-16-2411↗
- Thilo Hackert · 6 shared papers, 2021–2025 · strength 0.90
- Paper Perioperative immunotherapy for pancreatic cancer is on its way. DOI 10.21037/hbsn-21-238↗
- Paper Pancreatic Adenocarcinoma: Long-Term Outcomes of Adjuvant Therapy in the ESPAC4 Phase III Trial. DOI 10.1200/jco.24.01118↗
- Paper Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. DOI 10.3389/fcell.2023.1179559↗
- Xu Zhou · 5 shared papers, 2021–2024 · strength 0.73
- Paper Refining the Treatment of Pancreatic Cancer From Big Data to Improved Individual Survival. DOI 10.1093/function/zqad011↗
- Paper Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. DOI 10.3389/fcell.2023.1179559↗
- Paper Metavert synergises with standard cytotoxics in human PDAC organoids and is associated with transcriptomic signatures o… DOI 10.1016/j.tranon.2024.102109↗
- Simon T. Barry · 5 shared papers, 2016–2023 · strength 0.69
- Paper Modulation of Type I Interferon Responses to Influence Tumor-Immune Cross Talk in PDAC. DOI 10.3389/fcell.2022.816517↗
- Paper Deconstructing Pancreatic Cancer Using Next Generation-Omic Technologies-From Discovery to Knowledge-Guided Platforms f… DOI 10.3389/fcell.2021.795735↗
- Paper Persister cell phenotypes contribute to poor patient outcomes after neoadjuvant chemotherapy in PDAC. DOI 10.1038/s43018-023-00628-6↗
- Rita Teresa Lawlor · 6 shared papers, 2014–2025 · strength 0.68
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Expression of the membrane tetraspanin claudin 18 on cancer cells promotes T lymphocyte infiltration and antitumor immu… DOI 10.1016/j.immuni.2024.04.021↗
- Aldo Scarpa · 6 shared papers, 2014–2025 · strength 0.68
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Expression of the membrane tetraspanin claudin 18 on cancer cells promotes T lymphocyte infiltration and antitumor immu… DOI 10.1016/j.immuni.2024.04.021↗
- Susanne Roth · 4 shared papers, 2021–2024 · strength 0.65
- Paper Tumour plasticity and tumour microenvironment interactions as potential immunologic targets for pancreatic cancer treat… DOI 10.21037/cco-24-72↗
- Paper Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. DOI 10.3389/fcell.2023.1179559↗
- Paper Metavert synergises with standard cytotoxics in human PDAC organoids and is associated with transcriptomic signatures o… DOI 10.1016/j.tranon.2024.102109↗
- Christian P. Pilarsky · 5 shared papers, 2019–2025 · strength 0.64
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper CRISPR Cas9 in Pancreatic Cancer Research. DOI 10.3389/fcell.2019.00239↗
- Paper CDK7 inhibition augments response to multidrug chemotherapy in pancreatic cancer. DOI 10.1186/s13046-022-02443-w↗
- Vincenzo Corbo · 5 shared papers, 2014–2025 · strength 0.61
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Expression of the membrane tetraspanin claudin 18 on cancer cells promotes T lymphocyte infiltration and antitumor immu… DOI 10.1016/j.immuni.2024.04.021↗
- Jingyu An · 4 shared papers, 2021–2024 · strength 0.54
- Paper Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. DOI 10.3389/fcell.2023.1179559↗
- Paper Metavert synergises with standard cytotoxics in human PDAC organoids and is associated with transcriptomic signatures o… DOI 10.1016/j.tranon.2024.102109↗
- Paper Persister cell phenotypes contribute to poor patient outcomes after neoadjuvant chemotherapy in PDAC. DOI 10.1038/s43018-023-00628-6↗
- Vera Oliveira · 2 shared papers, 2026 · strength 0.49
- Paper The Role of Circulating Tumor DNA in Surgical Management of Pancreatic Cancer: Systematic Review and Meta-analysis. DOI 10.1097/sla.0000000000007036↗
- Paper Pancreatic Cancer Frequently Infiltrates the TRIANGLE Compartment: A Surgical Option to Prevent Local Recurrence. DOI 10.1097/as9.0000000000000672↗
- Gil Gonçalves · 2 shared papers, 2026 · strength 0.49
- Paper The Role of Circulating Tumor DNA in Surgical Management of Pancreatic Cancer: Systematic Review and Meta-analysis. DOI 10.1097/sla.0000000000007036↗
- Paper Pancreatic Cancer Frequently Infiltrates the TRIANGLE Compartment: A Surgical Option to Prevent Local Recurrence. DOI 10.1097/as9.0000000000000672↗
- Filipe C. Borges · 2 shared papers, 2026 · strength 0.49
- Paper The Role of Circulating Tumor DNA in Surgical Management of Pancreatic Cancer: Systematic Review and Meta-analysis. DOI 10.1097/sla.0000000000007036↗
- Paper Pancreatic Cancer Frequently Infiltrates the TRIANGLE Compartment: A Surgical Option to Prevent Local Recurrence. DOI 10.1097/as9.0000000000000672↗
- Daniel Schreyer · 3 shared papers, 2021–2025 · strength 0.48
- Paper Deconstructing Pancreatic Cancer Using Next Generation-Omic Technologies-From Discovery to Knowledge-Guided Platforms f… DOI 10.3389/fcell.2021.795735↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper CDK7 inhibition augments response to multidrug chemotherapy in pancreatic cancer. DOI 10.1186/s13046-022-02443-w↗
- Francesca Lupo · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Antonia Malinova · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Sabrina L. D’Agosto · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Lisa Veghini · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Elena Fiorini · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- David Arthur Tuveson · 3 shared papers, 2017–2025 · strength 0.38
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Recurrent noncoding regulatory mutations in pancreatic ductal adenocarcinoma. DOI 10.1038/ng.3861↗
- Maarten F. Bijlsma · 2 shared papers, 2018–2022 · strength 0.32
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.