ResearcherUniversity of VeronaActive in the last 2 years
Lisa Veghini
Matched on ORCID iD 0000-0003-3289-1189; OpenAlex author ID A5067619674. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 58
2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 2 papers · 2025: 2 papers · 2026: 3 papers
Papers 10
- Deciphering transcriptional plasticity in pancreatic ductal adenocarcinoma reveals alterations in sensory neuron innervation. Co-author DOI 10.1002/1878-0261.70233↗
- Estrogen Shapes Fibroblast States to Limit Pancreatic Cancer Aggressiveness. Co-author DOI 10.1158/0008-5472.can-25-4671↗
- Multinucleated Giant Cells in Human Pancreatic Cancer Are a Distinct Macrophage Population Undergoing a DNA Damage Response and Associated with an Aggressive T… Co-author DOI 10.1158/2326-6066.cir-25-1602↗
- MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. Co-author DOI 10.1038/s41586-025-08721-9↗
- SMAD4 and KRAS Status Shapes Cancer Cell-Stromal Cross-Talk and Therapeutic Response in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-2330↗
- Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. Co-author DOI 10.1038/s41467-024-54975-8↗
- Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. Co-author DOI 10.1136/gutjnl-2023-329807↗
- Clinical and Genomic Characterization of Pancreatic Ductal Adenocarcinoma with Signet-Ring/Poorly Cohesive Cells. Co-author DOI 10.1016/j.modpat.2023.100251↗
- Loss of FGFR4 promotes the malignant phenotype of PDAC. Co-author DOI 10.1038/s41388-022-02432-5↗
- Cell Lineage Infidelity in PDAC Progression and Therapy Resistance. First author DOI 10.3389/fcell.2021.795251↗
Closest collaborators
The 25 strongest of 58 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Vincenzo Corbo · 10 shared papers, 2021–2026 · strength 2.20
- Paper Estrogen Shapes Fibroblast States to Limit Pancreatic Cancer Aggressiveness. DOI 10.1158/0008-5472.can-25-4671↗
- Paper Deciphering transcriptional plasticity in pancreatic ductal adenocarcinoma reveals alterations in sensory neuron innerv… DOI 10.1002/1878-0261.70233↗
- Paper Multinucleated Giant Cells in Human Pancreatic Cancer Are a Distinct Macrophage Population Undergoing a DNA Damage Resp… DOI 10.1158/2326-6066.cir-25-1602↗
- Rita Teresa Lawlor · 5 shared papers, 2022–2025 · strength 0.71
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Aldo Scarpa Scarpa · 5 shared papers, 2022–2025 · strength 0.71
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Antonia Malinova · 4 shared papers, 2021–2025 · strength 0.62
- Paper Cell Lineage Infidelity in PDAC Progression and Therapy Resistance. DOI 10.3389/fcell.2021.795251↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Francesca Lupo · 4 shared papers, 2022–2025 · strength 0.59
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Sabrina L. D’Agosto · 4 shared papers, 2022–2025 · strength 0.59
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Elena Fiorini · 4 shared papers, 2022–2025 · strength 0.59
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Claudio Luchini · 4 shared papers, 2022–2025 · strength 0.56
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Clinical and Genomic Characterization of Pancreatic Ductal Adenocarcinoma with Signet-Ring/Poorly Cohesive Cells. DOI 10.1016/j.modpat.2023.100251↗
- Carmine Carbone · 3 shared papers, 2024–2025 · strength 0.48
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Peter J. Bailey · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Pietro Delfino · 3 shared papers, 2022–2024 · strength 0.42
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Michèle Milella · 3 shared papers, 2022–2025 · strength 0.40
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Clinical and Genomic Characterization of Pancreatic Ductal Adenocarcinoma with Signet-Ring/Poorly Cohesive Cells. DOI 10.1016/j.modpat.2023.100251↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Giulia Biffi · 2 shared papers, 2024–2025 · strength 0.36
- Antonio Agostini · 2 shared papers, 2024–2025 · strength 0.32
- David Arthur Tuveson · 2 shared papers, 2024–2025 · strength 0.32
- Geny Piro · 2 shared papers, 2024 · strength 0.31
- Giuseppe Malleo · 2 shared papers, 2023–2025 · strength 0.29
- Roberto Salvia · 2 shared papers, 2023–2025 · strength 0.29
- Paul A. Heppenstall · 1 shared paper, 2026 · strength 0.25
- Elena Cat Genova · 1 shared paper, 2026 · strength 0.25
- Antonio Pea · 2 shared papers, 2022–2023 · strength 0.23
- Giovanni Luigi Capretti · 1 shared paper, 2026 · strength 0.22
- Marika Viatore · 1 shared paper, 2026 · strength 0.22
- Marco Erreni · 1 shared paper, 2026 · strength 0.22
- Greta Donisi · 1 shared paper, 2026 · strength 0.22
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.