ResearcherUniversity of VeronaActive in the last 2 years
Elena Fiorini
Matched on ORCID iD 0009-0004-7423-8908; OpenAlex author ID A5063367641. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 43
2019: 1 paper · 2020: none · 2021: none · 2022: 1 paper · 2023: none · 2024: 2 papers · 2025: 2 papers · 2026: 1 paper
Papers 7
- Tumor-specific targeting of a mitochondrial Kv1.3 channel inhibitor through conjugation to gastrin/cholecystokinin B receptor ligand strongly reduces pancreati… Co-author DOI 10.1016/j.phrs.2026.108150↗
- MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. Co-author DOI 10.1038/s41586-025-08721-9↗
- A first-in-class inhibitor of homologous recombination DNA repair counteracts tumour growth, metastasis and therapeutic resistance in pancreatic cancer. Co-author DOI 10.1186/s13046-025-03389-5↗
- Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. Co-author DOI 10.1038/s41467-024-54975-8↗
- Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. Co-author DOI 10.1136/gutjnl-2023-329807↗
- Loss of FGFR4 promotes the malignant phenotype of PDAC. Co-author DOI 10.1038/s41388-022-02432-5↗
- MAP Kinase inhibition reshapes tumor microenvironment of mouse pancreatic cancer by depleting anti-inflammatory macrophages Co-author DOI 10.1101/759779↗
Closest collaborators
The 25 strongest of 43 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Vincenzo Corbo · 7 shared papers, 2019–2026 · strength 1.09
- Paper Tumor-specific targeting of a mitochondrial Kv1.3 channel inhibitor through conjugation to gastrin/cholecystokinin B re… DOI 10.1016/j.phrs.2026.108150↗
- Paper A first-in-class inhibitor of homologous recombination DNA repair counteracts tumour growth, metastasis and therapeutic… DOI 10.1186/s13046-025-03389-5↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Francesca Lupo · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Sabrina L. D’Agosto · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Rita Teresa Lawlor · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Aldo Scarpa Scarpa · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Lisa Veghini · 4 shared papers, 2022–2025 · strength 0.59
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Pietro Delfino · 4 shared papers, 2019–2024 · strength 0.49
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Carmine Carbone · 3 shared papers, 2024–2025 · strength 0.48
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Claudio Luchini · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Peter J. Bailey · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Antonia Malinova · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Antonio Agostini · 2 shared papers, 2024–2025 · strength 0.32
- David Arthur Tuveson · 2 shared papers, 2024–2025 · strength 0.32
- Geny Piro · 2 shared papers, 2024 · strength 0.31
- Michèle Milella · 2 shared papers, 2022–2025 · strength 0.28
- Sofia Parrasia · 1 shared paper, 2026 · strength 0.23
- Erich Gulbins · 1 shared paper, 2026 · strength 0.23
- Lucia Biasutto · 1 shared paper, 2026 · strength 0.23
- Alessandro Carrer · 1 shared paper, 2026 · strength 0.23
- Carlotta Paoli · 1 shared paper, 2026 · strength 0.23
- Ildikò Szabó · 1 shared paper, 2026 · strength 0.23
- Phyllis F. Cheung · 2 shared papers, 2019–2024 · strength 0.23
- Jens Thomas Siveke · 2 shared papers, 2019–2024 · strength 0.23
- Vincenzo Bronte · 2 shared papers, 2019–2024 · strength 0.22
- Francesco De Sanctis · 2 shared papers, 2019–2024 · strength 0.22
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.