ResearcherUniversity of VeronaActive in the last 2 years
Sabrina L. D’Agosto
Matched on ORCID iD 0000-0003-0950-9914; OpenAlex author ID A5029384948. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 37
2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: 2 papers · 2025: 1 paper
Papers 7
- MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. Co-author DOI 10.1038/s41586-025-08721-9↗
- Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. Co-author DOI 10.1038/s41467-024-54975-8↗
- Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. Co-author DOI 10.1136/gutjnl-2023-329807↗
- The tumor stroma influences immune cell distribution and recruitment in a PDAC-on-a-chip model. Co-author DOI 10.3389/fimmu.2023.1155085↗
- Loss of FGFR4 promotes the malignant phenotype of PDAC. Co-author DOI 10.1038/s41388-022-02432-5↗
- Combinatorial Effect of Magnetic Field and Radiotherapy in PDAC Organoids: A Pilot Study. Last author DOI 10.3390/biomedicines8120609↗
- MAP Kinase inhibition reshapes tumor microenvironment of mouse pancreatic cancer by depleting anti-inflammatory macrophages Co-author DOI 10.1101/759779↗
Closest collaborators
The 25 strongest of 37 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Vincenzo Corbo · 7 shared papers, 2019–2025 · strength 1.00
- Paper The tumor stroma influences immune cell distribution and recruitment in a PDAC-on-a-chip model. DOI 10.3389/fimmu.2023.1155085↗
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Rita Teresa Lawlor · 6 shared papers, 2019–2025 · strength 0.76
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Aldo Scarpa Scarpa · 6 shared papers, 2019–2025 · strength 0.76
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Francesca Lupo · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Elena Fiorini · 5 shared papers, 2019–2025 · strength 0.66
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Lisa Veghini · 4 shared papers, 2022–2025 · strength 0.59
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Pietro Delfino · 4 shared papers, 2019–2024 · strength 0.49
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Carmine Carbone · 3 shared papers, 2024–2025 · strength 0.48
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Claudio Luchini · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Differential activity of MAPK signalling defines fibroblast subtypes in pancreatic cancer. DOI 10.1038/s41467-024-54975-8↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Peter J. Bailey · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Antonia Malinova · 3 shared papers, 2022–2025 · strength 0.43
- Paper MYC ecDNA promotes intratumour heterogeneity and plasticity in PDAC. DOI 10.1038/s41586-025-08721-9↗
- Paper Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC. DOI 10.1136/gutjnl-2023-329807↗
- Paper Loss of FGFR4 promotes the malignant phenotype of PDAC. DOI 10.1038/s41388-022-02432-5↗
- Antonio Agostini · 2 shared papers, 2024–2025 · strength 0.32
- David Arthur Tuveson · 2 shared papers, 2024–2025 · strength 0.32
- Geny Piro · 2 shared papers, 2024 · strength 0.31
- Michèle Milella · 2 shared papers, 2022–2025 · strength 0.28
- Marlene Geyer · 1 shared paper, 2023 · strength 0.24
- Karla C. S. Queiroz · 1 shared paper, 2023 · strength 0.24
- Phyllis F. Cheung · 2 shared papers, 2019–2024 · strength 0.23
- Jens Thomas Siveke · 2 shared papers, 2019–2024 · strength 0.23
- Vincenzo Bronte · 2 shared papers, 2019–2024 · strength 0.22
- Francesco De Sanctis · 2 shared papers, 2019–2024 · strength 0.22
- Antonio Pea · 2 shared papers, 2020–2022 · strength 0.21
- Claudia Fiorini · 2 shared papers, 2019–2022 · strength 0.18
- Giuseppe Malleo · 1 shared paper, 2025 · strength 0.17
- Daniel Schreyer · 1 shared paper, 2025 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.